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Human and rat β cells differ in glucose transporter but not in glucokinase gene expression.

At the same time, another player – a team of researchers from Imperial College, London – identified an enzyme called glucokinase which could possibly be target for a new pill because it drives the craving for sugar in the brain.

His tests on rats showed that boosting glucokinase activity caused the animals to consume glucose over normal food.

When the researchers increased the activity of glucokinase in the hypothalamus using a virus, rats consumed more glucose in preference to chow.

When glucokinase activity was reduced, they consumed less glucose.