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By modulating the spatial distribution of myosin -- the molecular motor that drives cell movement -- the researchers were able to control the positioning of these attractors, directing cell accumulation to targeted areas of the embryo.

The guideline includes recommendations for adding cardiac myosin inhibitors, a new class of medication for patients with symptomatic obstructive HCM who do not get adequate symptom relief from first-line drug therapy.

The findings also suggest myosin -- a type of motor protein involved in muscle contraction -- plays a role in non-shivering thermogenesis during hibernation, where heat is produced independent of the muscle activity of shivering.

Active RhoA turns on the motor protein non-muscle myosin II, which causes the back ends of the cells to constrict and let go of the surface they are moving along.

The study titled "Cryo-EM structure of the human cardiac myosin filament" was published online in the journal Nature earlier this month.