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NMNAT2 is a vital provider of nicotinamide adenine dinucleotide for the brain.

Another drug candidate involves a coenzyme called nicotinamide adenine dinucleotide, or N.A.D.

Eliciting the mitochondrial unfolded protein response by nicotinamide adenine dinucleotide repletion reverses fatty liver disease in mice.

The process of borrowing a bacterial enzyme to control nicotinamide adenine dinucleotide concentrations in mammalian cells is discussed.

The process of borrowing a bacterial enzyme to control nicotinamide adenine dinucleotide concentrations in mammalian cells is discussed.