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Murach added, "The take-home isn't necessarily that we need to induce MYC in muscle to mimic exercise, but that we can harness the knowledge of what this oncogene affects in muscle and then try to design therapies and interventions for atrophy and enhancing muscle adaptability that activate those positive downstream effects of MYC without evoking the possibility of oncogenesis."

Even more surprisingly, further analyses revealed that genomic deletions might be a more widespread mechanism of oncogenesis and resistance than previously thought.

Zachary Schug, Ph.D., assistant professor in the Molecular and Cellular Oncogenesis Program of the Ellen and Ronald Caplan Cancer Center at The Wistar Institute, has published a new paper in the journal Nature Cancer.

Gacche has published in top journals, including Oncogenesis, a Nature publication.

“The reprogramming process and oncogenesis share several characteristics,” she says.