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“Unfortunately, we don’t know why. There is so much we don’t understand about the pathophysiology of what makes it more lethal for one animal than another.”

The study, which focuses on the role of reactive astrocytes and the plexin-B1 protein in Alzheimer's pathophysiology, provides crucial insights into brain cell communication and opens the door to innovative treatment strategies.

"This work advances understanding of schizophrenia pathophysiology at greater detail across both the complex landscape of cells within the brain, and the diverse experiences of people with this disease," said Ruzicka, who is also associate medical director of Harvard Brain Tissue Resource Center at McLean, and an assistant professor of Psychiatry at Harvard Medical School.

This includes a comprehensive understanding of the pathophysiology and early detection of those patients who have a high risk of an unfavorable outcome.

This observation advances molecular insights into the pathophysiology of adverse heat events and will serve as a foundation for future research.