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"And what the SSRIs tended to do is to regrow these neurons. But here was the rub: they did so on a timescale that correlated with their clinical efficacy, not with their ability to promote serotonin increases in the synaptic cleft."

This synaptic cleft is a narrow gap that is not easily accessible to an antibody.

Finger-like cellular processes extended from the cancer cells to reach existing mouse synapses between neurons, adopting the same typical position around a synapse as is adopted by glial cells that remove glutamate from the synaptic cleft.

Forging this network is a dynamic process, involving proteins at the synaptic cleft that function in the formation, maturation, and remodeling of these connections.